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How to customize Maven project for using OSGi?

I have a java project in which I used OSGi, but as I would like to start using maven, can someone give a tutorial to how to use maven to use OSGi?


Most of the answers to this question are many years old and some are not relevant now.
To use Maven as a build tool for any OSGi project, start with the maven-osgi-plugin documentation.
The latest version is available as a Maven artifact.
Next read about the concepts, the capabilities and the use cases of the maven-enforcer-plugin
The maven-bundle-plugin is not yet compatible with OSGi but is very simple.
For further reference of the OSGi ecosystem you should use the Eclipse WTP.


To use OSGi with Maven you need to install the Maven Fuse plugin.
Maven Fuse is an Eclipse plugin that provides a unified view of your projects. Here’s how you install it on your Eclipse workspace:
Once you have Maven Fuse installed, you can view your OSGi projects in a unified window:


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const things = [{name:’something’, age: 12}, {name:’something’, age: 15}]

function getRandomObject(things, index) {
const randomThing = things[Math.floor(Math.random()

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In order to maintain self-renewal and homeostasis of adult tissues, mechanisms must exist to prevent proliferation of cells that lack a normal complement of stem cell factors. We have previously shown that the p19(ARF) protein is such a tumor suppressor in the hematopoietic system. In studies presented in this paper, we show that p19(ARF) is also required for complete tumor suppression in other cell types. This is evidenced by the finding that ectopic expression of p19(ARF) leads to an inhibition of the growth of transformed epithelial cell lines. In contrast to the hematopoietic system, in these cells p19(ARF) does not appear to be involved in the down-regulation of cyclin D/cdk complexes. In addition, p19(ARF)-deficient fibroblasts are not able to activate wild-type p53. These results identify p19(ARF) as a potent tumor suppressor protein that is required not only for the self-renewal of adult tissues but also for complete tumor suppression.The role of the spin-orbit coupling constant B in electron and hole concentration and charge rearrangement in semiconductor nanocrystals.
An exact and simple theoretical model is given in order to analyze the role of the spin-orbit coupling constant B in the electron and hole concentration and charge rearrangement in nanocrystalline semiconductors. The model is used for interpretation of data obtained in a recent electron paramagnetic resonance and photoluminescence study on CdSe nanoparticles. The model takes into account the important interactions between the conduction band states, the valence band states, and phonons of the host matrix, in contrast to previous studies in which the interactions between the valence band and con